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Volume 23, Number 5—May 2017
Dispatch

Exposure Risk for Infection and Lack of Human-to-Human Transmission of Mycobacterium ulcerans Disease, Australia

Daniel P. O’BrienComments to Author , James W. Wynne, Andrew H. Buultjens, Wojtek P. Michalski, Timothy P. Stinear, N. Deborah Friedman, Andrew Hughes, and Eugene Athan
Author affiliations: Médecins Sans Frontières, London, UK (D.P. O’Brien); University of Melbourne, Melbourne, Victoria, Australia (D.P. O’Brien, A.H. Buultjens, T.P. Stinear); Barwon Health, Geelong, Victoria, Australia (D.P. O’Brien, N.D. Friedman, A. Hughes, E. Athan); Commonwealth Scientific and Industrial Research Organisation, Geelong (J.W. Wynne, W.P. Michalski); Deakin University School of Medicine, Geelong (E. Athan)

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Figure 2

Median joining network of 12 SNPs of 12 Mycobacterium ulcerans isolates from patients with Mycobacterium ulcerans disease, Barwon Health cohort, Bellarine Peninsula, Victoria, Australia, 1998–2016. Node colors indicate clusters. Blue, cluster 4a/4b; red, cluster 1a/1b; green, cluster 2a/2b. Black nodes represent 7 unrelated isolates. The size of each node is proportional to the number of genetically identical isolates with identical genotypes. Values indicate number of SNPs between each node. SN

Figure 2. Median joining network of 12 SNPs of 12 Mycobacterium ulcerans isolates from patients with Mycobacterium ulcerans disease, Barwon Health cohort, Bellarine Peninsula, Victoria, Australia, 1998–2016. Node colors indicate clusters. Blue, cluster 4a/4b; red, cluster 1a/1b; green, cluster 2a/2b. Black nodes represent 6 unrelated isolates. The size of each node is proportional to the number of genetically identical isolates with identical genotypes. Values indicate number of SNPs between each node. SNP, single-nucleotide polymorphism.

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Page created: April 14, 2017
Page updated: April 14, 2017
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