Human Infection with Highly Pathogenic Avian Influenza A(H7N9) Virus, China
, Chris Ka Pun Mok1
, Wenfei Zhu1
, Haibo Zhou1
, Jianfeng He, Wenda Guan, Jie Wu, Wenjun Song, Dayan Wang, Jiexiong Liu, Qinhan Lin, Daniel Ka Wing Chu, Lei Yang, Nanshan Zhong, Zifeng Yang2
, Yuelong Shu2
, and Joseph Sriyal Malik Peiris2
Author affiliations: Guangdong Provincial Center for Disease Control and Prevention, Guangzhou, China (C. Ke, J. He, J. Wu); First Affiliated Hospital of Guangzhou Medical University, State Key Laboratory of Respiratory Disease, Guangzhou (C.K.P. Mok, W. Guan, D.K.W. Chu, N. Zhong, Z. Yang, J.S.M. Peiris); The University of Hong Kong, Hong Kong, China (C.K.P. Mok, J.S.M. Peiris); National Institute for Viral Disease Control and Prevention, China CDC, Beijing, China (W. Zhu, D. Wang, L. Yang, Y. Shu); The Sixth Affiliated Hospital of Guangzhou Medical University, Qingyuan, China (H. Zhou, J. Liu, Q. Lin); Jinan University, Guangzhou (W. Song); Macau University of Science and Technology, Macau, China (N. Zhong, Z. Yang)
Figure 3. Kinetics of viral load, oxygenation index, and HI antibody titers in 56-year-old man infected with highly pathogenic avian influenza A(H7N9) virus, China, 2017. Arrows indicate the days HI titers and viral titers in serum were acquired. HI, hemagglutination inhibition; neg, negative.
Page created: July 18, 2017
Page updated: July 18, 2017
Page reviewed: July 18, 2017
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