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Volume 25, Number 3—March 2019
Synopsis

Cross-Border Movement of Highly Drug-Resistant Mycobacterium tuberculosis from Papua New Guinea to Australia through Torres Strait Protected Zone, 2010–2015

Arnold Bainomugisa, Sushil Pandey, Ellen Donnan, Graham Simpson, J’Belle Foster, Evelyn Lavu, Stenard Hiasihri, Emma S. McBryde, Rendi Moke, Steven Vincent, Vitali Sintchenko, Ben J. Marais, Lachlan J.M. Coin, and Christopher CoulterComments to Author 
Author affiliations: Pathology Queensland, Brisbane, Queensland, Australia (A. Bainomugisa, S. Pandey, C. Coulter); The University of Queensland, Brisbane (A. Bainomugisa, L.J.M. Coin); Queensland Department of Health, Brisbane (E. Donnan, C. Coulter); Cairns Tuberculosis Unit, Cairns, Queensland, Australia (G. Simpson, S. Vincent); James Cook University, Townsville, Queensland, Australia (J. Foster, E.S. McBryde); Central Public Health Laboratory, Port Moresby, Papua New Guinea (E. Lavu); Western Province Health Office, Daru, Papua New Guinea (S. Hiasihri); Port Moresby General Hospital, Port Moresby (R. Moke); Westmead Hospital, Sydney, New South Wales, Australia (V. Sintchenko); The University of Sydney, Sydney (V. Sintchenko, B.J. Marais)

Main Article

Figure 4

Correlation of phenotypic and genotypic drug resistance among modern Beijing isolates from Papua New Guinea and Australia citizens residing or previously residing in the Torres Strait Protected Zone, 2010–2015. No resistance mutation detected in 1 kanamycin-resistant strain, despite targeted sequencing of rrs and eis genes. *Pairs of co-occurring mutations observed within the same isolates were embA-embB (C12T) with embB (p.Asp354Ala) and embB (p.Gly406Val) with embB (p.Met306Ile). AMGs, aminogl

Figure 4. Correlation of phenotypic and genotypic drug resistance among modern Beijing isolates from Papua New Guinea and Australia citizens residing or previously residing in the Torres Strait Protected Zone, 2010–2015. No resistance mutation detected in 1 kanamycin-resistant strain, despite targeted sequencing of rrs and eis genes. *Pairs of co-occurring mutations observed within the same isolates were embA-embB (C12T) with embB (p.Asp354Ala) and embB (p.Gly406Val) with embB (p.Met306Ile). AMGs, aminoglycosides; EMB, ethambutol; ETA, ethionamide; INH, isoniazid; PZA, pyrazinamide; RIF, rifampin; STR, streptomycin.

Main Article

Page created: February 19, 2019
Page updated: February 19, 2019
Page reviewed: February 19, 2019
The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
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