Plasmodium falciparum pfhrp2 and pfhrp3 Gene Deletions and Relatedness to Other Global Isolates, Djibouti, 2019–2020
Eric Rogier
1 , Jessica N. McCaffery
1, Mohamed Ali Mohamed, Camelia Herman, Doug Nace, Rachel Daniels, Naomi Lucchi, Sophie Jones, Ira Goldman, Michael Aidoo, Qin Cheng, Edie A. Kemenang, Venkatachalam Udhayakumar, and Jane Cunningham
Author affiliations: Centers for Disease Control and Prevention, Atlanta, Georgia, USA (E. Rogier, J.N. McCaffery, C. Herman, D. Nace, N. Lucchi, S. Jones, I. Goldman, M. Aidoo, V. Udhayakumar); Oak Ridge Institute for Science and Education, Oak Ridge, Tennessee, USA (J.N. McCaffery); Hôpital Général Peltier, Djibouti City, Djibouti (M.A. Mohamed); Harvard T.H. Chan School of Public Health, Boston, Massachusetts, USA (R. Daniels); Broad Institute, Cambridge, Massachusetts, USA (R. Daniels); Australian Defence Force Malaria and Infectious Disease Institute, Brisbane, Queensland, Australia (Q. Cheng); World Health Organization, Geneva, Switzerland (E.A. Kemenang, J. Cunningham)
Main Article
Figure 3
Figure 3. Relatedness of Plasmodium falciparum parasites from Djibouti, 2019–2020, with different pfhrp2 and pfhrp3 genotypes. Cluster PC analysis shown for 7 neutral microsatellite data for monogenomic infections by subpopulations: pfhrp2+/pfhrp3+ (n = 16), pfhrp2+/pfhrp3– (n = 15), pfhrp2–/pfhrp3+ (n = 4), pfhrp2–/pfhrp3– (n = 17). Plot shown with PC1 on x-axis and PC2 on y-axis with 95% confidence ellipses. PC, principal component.
Main Article
Page created: August 11, 2022
Page updated: September 20, 2022
Page reviewed: September 20, 2022
The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.