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Volume 17, Number 8—August 2011
Research

Seroprevalence of Trichodysplasia Spinulosa–associated Polyomavirus

Els van der MeijdenComments to Author , Siamaque Kazem, Manda M. Burgers, Rene Janssens, Jan Nico Bouwes Bavinck, Hester E. de Melker, and Mariet C.W. Feltkamp
Author affiliations: Author affiliations: Leiden University Medical Center, Leiden, the Netherlands (E. van der Meijden, S. Kazem, M.M. Burgers, J.N. Bouwes Bavinck, M.C.W. Feltkamp); Jeroen Bosch Ziekenhuis, ‘s Hertogenbosch, the Netherlands (R. Janssens); National Institute of Public Health and the Environment, Bilthoven, the Netherlands (H. de Melker)

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Figure 5

Seroresponses against trichodysplasia spinulosa–associated polyomavirus (TSV) (A) and BKV polyomavirus (B) for a patient with trichodysplasia spinulosa, the Netherlands. Serial dilutions of serum from a TS patient were tested for reactivity against TSV viral protein 1 (VP1) or BKV VP1 by using the VP1 multiplex antibody-binding assay. Samples were preincubated with soluble recombinant glutathione-S-transferase (GST) (black line), GST-BKV VP1 (red line), or GST-TSV VP1 (blue line). MFI, median fl

Figure 5. Seroresponses against trichodysplasia spinulosa–associated polyomavirus (TSV) (A) and BKV polyomavirus (B) for a patient with trichodysplasia spinulosa, the Netherlands. Serial dilutions of serum from a TS patient were tested for reactivity against TSV viral protein 1 (VP1) or BKV VP1 by using the VP1 multiplex antibody-binding assay. Samples were preincubated with soluble recombinant glutathione-S-transferase (GST) (black line), GST-BKV VP1 (red line), or GST-TSV VP1 (blue line). MFI, median fluorescent intensity.

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Page created: August 15, 2011
Page updated: August 15, 2011
Page reviewed: August 15, 2011
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