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Volume 20, Number 9—September 2014
Synopsis

Encephalitis Caused by Pathogens Transmitted through Organ Transplants, United States, 2002–2013

Sridhar V. BasavarajuComments to Author , Matthew J. Kuehnert, Sherif Zaki, and James Sejvar
Author affiliations: Centers for Disease Control and Prevention, Atlanta, Georgia, USA

Main Article

Figure 3

Photomicrographs showing histopathologic features and immunolocalization of lymphocytic choriomeningitis virus (LCMV) antigens in liver tissue from a liver transplant recipient with donor-derived LCMV infection. A) Massive hepatic necrosis, without prominent inflammation. Hematoxylin and eosin staining. Original magnification, ×50. B) LCMV antigens within hepatocytes and sinusoidal lining cells. Immunoalkaline phosphate staining, naphthol fast red substrate with light hematoxylin counterstain. O

Figure 3. Photomicrographs showing histopathologic features and immunolocalization of lymphocytic choriomeningitis virus (LCMV) antigens in liver tissue from a liver transplant recipient with donor-derived LCMV infection. A) Massive hepatic necrosis, without prominent inflammation. Hematoxylin and eosin staining. Original magnification, ×50. B) LCMV antigens within hepatocytes and sinusoidal lining cells. Immunoalkaline phosphate staining, naphthol fast red substrate with light hematoxylin counterstain. Original magnification, ×158.

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Page created: August 13, 2014
Page updated: August 13, 2014
Page reviewed: August 13, 2014
The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
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