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Volume 27, Number 4—April 2021

Dispatch

Rare Norovirus GIV Foodborne Outbreak, Wisconsin, USA

Leslie BarclayComments to Author , Tim Davis, and Jan Vinjé
Author affiliations: Centers for Disease Control and Prevention, Atlanta, Georgia, USA (L. Barclay, J. Vinjé); Wisconsin State Laboratory of Hygiene, Madison, Wisconsin, USA (T. Davis)

Main Article

Figure 2

Maximum-likelihood phylogenetic analysis of rare norovirus GIV isolated during foodborne outbreak, Wisconsin, USA (red text), and reference strains. A) Partial polymerase gene (762 nt); B) complete capsid (VP1) gene (554 aa). Bootstrap support for 500 replicates is indicated on branches. For polymerase analysis, evolutionary distances were inferred by the Tamura-Nei model. For VP1 analysis, evolutionary distances were inferred by using the Jones-Taylor-Thornton matrix-based model. Reference strains are represented by type and GenBank accession number. Scale bar in panel A indicates nucleotide substitutions per site, and scale bar in panel B indicates amino acid substitutions per site.

Figure 2. Maximum-likelihood phylogenetic analysis of rare norovirus GIV isolated during foodborne outbreak, Wisconsin, USA (red text), and reference strains. A) Partial polymerase gene (762 nt); B) complete capsid (VP1) gene (554 aa). Bootstrap support for 500 replicates is indicated on branches. For polymerase analysis, evolutionary distances were inferred by the Tamura-Nei model. For VP1 analysis, evolutionary distances were inferred by using the Jones-Taylor-Thornton matrix-based model. Reference strains are represented by type and GenBank accession number. Scale bar in panel A indicates nucleotide substitutions per site, and scale bar in panel B indicates amino acid substitutions per site.

Main Article

Page created: January 19, 2021
Page updated: March 18, 2021
Page reviewed: March 18, 2021
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