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Volume 28, Number 4—April 2022
CME ACTIVITY - Research

Unique Clinical, Immune, and Genetic Signature in Patients with Borrelial Meningoradiculoneuritis1

Katarina Ogrinc2, Sergio A. Hernández2, Miša Korva, Petra Bogovič, Tereza Rojko, Lara Lusa, Geena Chiumento, Franc Strle3, and Klemen Strle3Comments to Author 
Author affiliations: University Medical Center Ljubljana, Ljubljana, Slovenia (K. Ogrinc, P. Bogovič, T. Rojko, F. Strle); New York State Department of Health, Albany, New York, USA (S.A. Hernández, K. Strle); University of Ljubljana, Ljubljana (M. Korva, L. Lusa); Massachusetts Department of Public Health, Boston, Massachusetts, USA (G. Chiumento)

Main Article

Figure 4

Correlation between the levels of 5 inflammatory mediators in CSF and CSF leukocyte counts among 120 patients with Lyme neuroborreliosis treated in Ljubljana, Slovenia, during 2006–2013. A) CXCL9; B) CXCL10; C) IL-17; D) CXCL12; E) CXCL13. Solid lines inside the graph represent linear regression; dotted lines indicate 95% CIs. ρ and p values were derived using the Spearman correlation. CSF, cerebrospinal fluid.

Figure 4. Correlation between the levels of 5 inflammatory mediators in CSF and CSF leukocyte counts among 120 patients with Lyme neuroborreliosis treated in Ljubljana, Slovenia, during 2006–2013. A) CXCL9; B) CXCL10; C) IL-17; D) CXCL12; E) CXCL13. Solid lines inside the graph represent linear regression; dotted lines indicate 95% CIs. ρ and p values were derived using the Spearman correlation. CSF, cerebrospinal fluid.

Main Article

1Part of the research reported here was presented at the 31st European Congress of Clinical Microbiology & Infectious Diseases (online), July 9–12, 2021.

2These first authors contributed equally to this article.

3These senior authors contributed equally to this article.

Page created: February 22, 2022
Page updated: March 22, 2022
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