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Volume 28, Number 4—April 2022
CME ACTIVITY - Research

Unique Clinical, Immune, and Genetic Signature in Patients with Borrelial Meningoradiculoneuritis1

Katarina Ogrinc2, Sergio A. Hernández2, Miša Korva, Petra Bogovič, Tereza Rojko, Lara Lusa, Geena Chiumento, Franc Strle3, and Klemen Strle3Comments to Author 
Author affiliations: University Medical Center Ljubljana, Ljubljana, Slovenia (K. Ogrinc, P. Bogovič, T. Rojko, F. Strle); New York State Department of Health, Albany, New York, USA (S.A. Hernández, K. Strle); University of Ljubljana, Ljubljana (M. Korva, L. Lusa); Massachusetts Department of Public Health, Boston, Massachusetts, USA (G. Chiumento)

Main Article

Figure 6

Frequency of TLR1–1805 genotypes, stratified by clinical manifestations (meningoradiculoneuritis and PFP/meningitis) among patients with Lyme neuroborreliosis treated in Ljubljana, Slovenia, during 2006–2013. The frequency of TLR1–1805 genotypes was determined via PCR restriction fragment-length polymorphism. Statistical analyses were performed using Fisher exact test for categorical variables or odds ratios. PFP, peripheral facial palsy; SNP, single-nucleotide polymorphism.

Figure 6. Frequency of TLR1–1805 genotypes, stratified by clinical manifestations (meningoradiculoneuritis and PFP/meningitis) among patients with Lyme neuroborreliosis treated in Ljubljana, Slovenia, during 2006–2013. The frequency of TLR1–1805 genotypes was determined via PCR restriction fragment-length polymorphism. Statistical analyses were performed using Fisher exact test for categorical variables or odds ratios. PFP, peripheral facial palsy; SNP, single-nucleotide polymorphism.

Main Article

1Part of the research reported here was presented at the 31st European Congress of Clinical Microbiology & Infectious Diseases (online), July 9–12, 2021.

2These first authors contributed equally to this article.

3These senior authors contributed equally to this article.

Page created: February 22, 2022
Page updated: March 22, 2022
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