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Volume 32, Number 4—April 2026

Dispatch

Cardiomyopathy Caused by Coxsackievirus Strain A9 in Previously Healthy Child, Northeastern France, 2024

Anne-Laure Lebreil1, Maxime Bisseux1, Audrey Mirand, Marie Glenet, Yohan N’Guyen, Norhan Taha, Pierre Mauran, Cecile Henquell, and Laurent AndreolettiComments to Author 
Author affiliation: INSERM UMR-S 1320, University of Reims Champagne-Ardenne, Reims, France (A.-L. Lebreil, M. Glenet, Y. N’Guyen, L. Andreoletti); National Reference Center for Enteroviruses and Parechoviruses, CHU Clermont-Ferrand, Clermont-Ferrand, France (M. Bisseux, A. Mirand, C. Henquell); UMR CNRS 6023, Université Clermont Auvergne, Clermont-Ferrand (M. Bisseux, A. Mirand, C. Henquell); University Hospital Centre of Reims, Reims (Y. N’Guyen, L. Andreoletti); American Memorial Hospital, Reims University Hospital, Reims (N. Taha, P. Mauran).

Main Article

Table

Laboratory tests performed on a child with recombinant mosaic coxsackievirus strain A9, associated with severe inflammatory cardiomyopathy, northeastern France, 2024*

Laboratory tests Day 1 Day 19 Day 97 Day 179
LVTDD, mm 47.1 (TTE) 47.6 (MRI) 34.4 (TTE)
T troponin, ng/L 57 41.3 12
NT ProBNP, pg/mL 32,843 2,897 251

*Corticosteroids plus intravenous Ig were administered on day 2, day 7, and day 16 of hospitalization. LVTDD, left ventricular tele diastolic diameters; MRI, magnetic resonance imaging; NT Pro BNP, N-terminal prohormone of brain natriuretic peptide; TTE, transthoracic echocardiography.

Main Article

1These authors contributed equally to this article.

Page created: March 12, 2026
Page updated: April 15, 2026
Page reviewed: April 15, 2026
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