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Volume 20, Number 12—December 2014
Research

Geographic Divergence of Bovine and Human Shiga Toxin–Producing Escherichia coli O157:H7 Genotypes, New Zealand1

Patricia JarosComments to Author , Adrian L. Cookson, Donald M. Campbell, Gail E. Duncan, Deborah Prattley, Philip E. Carter, Thomas E. Besser, Smriti Shringi, Steve Hathaway, Jonathan C. Marshall, and Nigel P. French
Author affiliations: Massey University, Palmerston North, New Zealand (P. Jaros, D. Prattley, J.C. Marshall, N.P. French); AgResearch Ltd, Palmerston North (A.L. Cookson); Ministry for Primary Industries, Wellington, New Zealand (D.M. Campbell, G.E. Duncan, S. Hathaway); Institute of Environmental Science and Research Ltd, Porirua, New Zealand (P. Carter); Washington State University, Pullman, Washington, USA (T.E. Besser, S. Shringi)

Main Article

Figure 1

Proportional distributions, stratified by year, of Shiga toxin–encoding bacteriophage insertion (SBI) types AY2a, WY12a, and ASY2c/SY2c of 363 human Shiga toxin–producing Escherichia coli O157:H7 isolates from clinical case-patients in New Zealand, 2008–2011. Error bars indicate 95% CIs.

Figure 1. Proportional distributions, stratified by year, of Shiga toxin–encoding bacteriophage insertion (SBI) types AY2a, WY12a, and ASY2c/SY2c of 363 human Shiga toxin–producing Escherichia coli O157:H7 isolates from clinical case-patients in New Zealand, 2008–2011. Error bars indicate 95% CIs.

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Main Article

1Preliminary results from this study were presented at the New Zealand Veterinary Association Conference; June 16–20, 2014, Hamilton, New Zealand.

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Page updated: November 18, 2014
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