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Volume 23, Number 12—December 2017
Synopsis

Spread of Canine Influenza A(H3N2) Virus, United States

Ian E.H. Voorhees, Amy L. Glaser, Kathy L. Toohey-Kurth, Sandra Newbury, Benjamin D. Dalziel, Edward Dubovi, Keith Poulsen, Christian Leutenegger, Katriina J.E. Willgert, Laura Brisbane-Cohen, Jill Richardson-Lopez, Edward C. Holmes, and Colin R. ParrishComments to Author 
Author affiliations: Cornell University, Ithaca, New York, USA (I.E.H. Voorhees, A.L. Glaser, E.J. Dubovi, K.J.E. Willgert, L. Brisbane-Cohen, C.R. Parrish); University of Wisconsin, Madison, Wisconsin, USA (K. Toohey-Kurth, S. Newbury, K. Poulsen); Oregon State University, Corvallis, Oregon, USA (B.D. Dalziel); IDEXX Laboratories, West Sacramento, California, USA (C. Leutenegger); Royal Veterinary College, London, UK (K.J.E. Willgert); Merck Animal Health, Madison, New Jersey, USA (J. Richardson-Lopez); University of Sydney, Sydney, New South Wales, Australia (E.C. Holmes)

Main Article

Figure 3

Distribution of clinical samples testing positive (red dots) and negative (blue dots) for canine influenza A(H3N2) virus RNA, United States, March–December 2015.

Figure 3. Distribution of clinical samples testing positive (red dots) and negative (blue dots) for canine influenza A(H3N2) virus RNA, United States, March–December 2015.

Main Article

Page created: November 16, 2017
Page updated: November 16, 2017
Page reviewed: November 16, 2017
The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
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