Virulence and Evolution of West Nile Virus, Australia, 1960–2012
Natalie A. Prow
12, Judith H. Edmonds
2, David T. Williams, Yin X. Setoh, Helle Bielefeldt-Ohmann, Willy W. Suen, Jody Hobson-Peters, Andrew F. van den Hurk, Alyssa T. Pyke, Sonja Hall-Mendelin, Judith A. Northill, Cheryl A. Johansen, David Warrilow, Jianning Wang, Peter D. Kirkland, Stephen Doggett, Christy C. Andrade
3, Aaron C. Brault, Alexander A. Khromykh
4 , and Roy A. Hall
4
Author affiliations: The University of Queensland, St. Lucia, Queensland, Australia (N.A. Prow, J.H. Edmonds, Y.X. Setoh, H. Bielefeldt-Ohmann, W.W. Suen, J. Hobson-Peters, A.A. Khromykh, R.A. Hall); CSIRO Australian Animal Health Laboratory, Geelong, Victoria, Australia (D.T. Williams, J. Wang); The University of Queensland, Gatton, Queensland, Australia (H. Bielefeldt-Ohmann, W.W. Suen); Department of Health, Brisbane, Queensland, Australia (A.F. van den Hurk, A.T. Pyke, S. Hall-Mendelin, J.A. Northill, D. Warrilow); The University of Western Australia, Nedlands, Western Australia, Australia (C.A. Johansen); Elizabeth Macarthur Agriculture Institute, Menangle, New South Wales, Australia (P.D. Kirkland); University of Sydney and Pathology West–ICPMR, Westmead, New South Wales, Australia (S. Doggett); University of California, Davis, California, USA (C.C. Andrade, A.C. Brault); Centers for Disease Control and Prevention, Fort Collins, Colorado, USA (C.C. Andrade, A.C. Brault)
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Figure 1
Figure 1. Growth kinetics of West Nile virus strains isolated in Australia, 1960–2012, in Vero (A) and C6/36 (B) cells. Cells were infected with a multiplicity of infection of 1, and the virus titers in the supernatants were determined by plaque assay on Vero cells.
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